Publications
RecA4142 causes SOS constitutive expression by loading onto reversed replication forks in Escherichia coli K-12.. J Bacteriol. 192(10):2575-82.
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2010. .
2009. .
2009.
Differential requirements of two recA mutants for constitutive SOS expression in Escherichia coli K-12.. PLoS One. 3(12):e4100.
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2008. RecA-mediated SOS induction requires an extended filament conformation but no ATP hydrolysis.. Mol Microbiol. 69(5):1165-79.
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2008. XthA (Exonuclease III) regulates loading of RecA onto DNA substrates in log phase Escherichia coli cells.. Mol Microbiol. 67(1):88-101.
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2008. .
2005.
Measurement of SOS expression in individual Escherichia coli K-12 cells using fluorescence microscopy.. Mol Microbiol. 53(5):1343-57.
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2004. Effects of mutations involving cell division, recombination, and chromosome dimer resolution on a priA2::kan mutant.. Proc Natl Acad Sci U S A. 98(15):8203-10.
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2001. PriA mutations that affect PriA-PriC function during replication restart.. Mol Microbiol. 41(3):697-704.
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2001. The importance of repairing stalled replication forks.. Nature. 404(6773):37-41.
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2000. .
1999.
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1996.
recO and recR mutations delay induction of the SOS response in Escherichia coli.. Mol Gen Genet. 246(2):254-8.
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1995. RecOR suppression of recF mutant phenotypes in Escherichia coli K-12.. J Bacteriol. 176(12):3661-72.
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1994. Studies on the mechanism of reduction of UV-inducible sulAp expression by recF overexpression in Escherichia coli K-12.. Mol Gen Genet. 245(6):741-9.
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1994.
Department of Microbiology