Publications
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1992. Class I major histocompatibility proteins are an essential component of the simian virus 40 receptor.. J Virol. 66(4):2037-45.
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1996. Bound simian virus 40 translocates to caveolin-enriched membrane domains, and its entry is inhibited by drugs that selectively disrupt caveolae.. Mol Biol Cell. 7(11):1825-34.
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1996. Extracellular simian virus 40 induces an ERK/MAP kinase-independent signalling pathway that activates primary response genes and promotes virus entry.. J Gen Virol. 77 ( Pt 9):2173-82.
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1998. MHC class I molecules are enriched in caveolae but do not enter with simian virus 40.. J Gen Virol. 79 ( Pt 6):1469-77.
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1999. Extracellular simian virus 40 transmits a signal that promotes virus enclosure within caveolae.. Exp Cell Res. 246(1):83-90.
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2001. Association of caveolin with Chlamydia trachomatis inclusions at early and late stages of infection.. Exp Cell Res. 266(2):229-38.
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2001. Replication of the gonad-specific virus Hz-2V in Ld652Y cells mimics replication in vivo.. J Invertebr Pathol. 77(1):44-50.
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2003. Trypanosoma brucei has two distinct mitochondrial DNA polymerase beta enzymes.. J Biol Chem. 278(49):49095-101.
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2006. Simian virus 40 late proteins possess lytic properties that render them capable of permeabilizing cellular membranes.. J Virol. 80(13):6575-87.
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2012. Disassembly of simian virus 40 during passage through the endoplasmic reticulum and in the cytoplasm.. J Virol. 86(3):1555-62.
Department of Microbiology